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dc.contributor.authorGarg, H.
dc.contributor.authorThompson, B.T.
dc.contributor.authorLinhardt, Robert J.
dc.contributor.authorHales, C.A.
dc.identifier.citationHeparin as a Potential Therapeutic Agent to Reverse Vascular Remodeling, H. Garg, B.T. Thompson, R.J. Linhardt, C.A. Hales, Proteoglycans and Lung Disease, H.G. Garg, P.J. Roughley, C.A. Hales (Eds.), Chapter 18, 377-398, 2003.
dc.descriptionProteoglycans and Lung Disease, H.G. Garg, P.J. Roughley, C.A. Hales (Eds.), Chapter 18, 377-398
dc.descriptionNote : if this item contains full text it may be a preprint, author manuscript, or a Gold OA copy that permits redistribution with a license such as CC BY. The final version is available through the publisher’s platform.
dc.description.abstractVascular changes after chronic hypoxia are characterized by hyperplasia, hypertrophy, and migration of smooth muscle cells (SMCs) in the media of muscular and partially muscular pulmonary arteries (1,2). A number of factors are known to cause SMC migration, such as serum, platelet-derived growth factor (PDGF)- BB (3,4), transforming growth factor-β (5), fibrinogen (6), oxidized low-density lipoprotein (7,8), and angiotensin II (9,10).
dc.relation.ispartofThe Linhardt Research Labs Online Collection
dc.relation.ispartofRensselaer Polytechnic Institute, Troy, NY
dc.subjectChemistry and chemical biology
dc.subjectChemical and biological engineering
dc.subjectBiomedical engineering
dc.titleHeparin as a Potential Therapeutic Agent to Reverse Vascular Remodeling
dc.typeBook chapter
dc.rights.holderIn Copyright : this Item is protected by copyright and/or related rights. You are free to use this Item in any way that is permitted by the copyright and related rights legislation that applies to your use. For other uses you need to obtain permission from the rights-holder(s).
dc.relation.departmentThe Linhardt Research Labs.
dc.relation.departmentThe Shirley Ann Jackson, Ph.D. Center for Biotechnology and Interdisciplinary Studies (CBIS)

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